Epidemiology and public health
RESEARCH ARTICLE   (Open Access)

Public Health Relevance of High Viral Load as a Risk Factor for Hepatocellular Carcinoma: A Bangladesh-Based Evaluation.

Israt Jahan Nisha1*, Mahathir Rahman Rudra2, Nafisa Binte Anower2

+ Author Affiliations

Clinical Epidemiology & Public Health 3(1) 1-9 https://doi.org/10.25163/health.3110265

Submitted: 06 March 2025  Revised: 09 May 2025  Published: 13 May 2025 

Abstract

Background: Hepatocellular carcinoma (HCC) is a widespread health matter and one of the top cancers assassination people, especially where there is a tall prevalence of infection with hepatitis B virus (HBV) and hepatitis C virus (HCV). Methods: This study recorded viral load levels, ALT, degree of fibrosis, cirrhosis, status of antiviral treatment and diagnosis of HBCC. With regard to these criteria, viral quantification for these patients was done with PCR standard procedures. Patients were stratified by thresholds of viral load, and the rest of the data was analyzed by using multivariate regression and survival analysis. Results: There was a distinct distinction in the progression of HCC across the patients; 18.5% of the HBV group and 9.7% of the HCV group with a high viral load progressed to HCC. On the other hand, patients on antiviral therapy had a significantly lower HCC incidence of 4.2% compared to 15.6% in those not receiving treatment. The highest identification rate (91.1%) for HCC was noted in patients monitored with comprehensive surveillance strategies including viral load testing. Conclusion: High viral load is one of the best predictors for the development of HCC because it strongly influences the manifestation of the disease; with proactive steps, better outcomes can be achieved. The risks associated with developing liver cancer are particularly important in understanding the need for implementing viral load testing as part of screening programs in the public health system of Bangladesh.

Keywords: Hepatocellular carcinoma, viral load, HBV, HCV, surveillance, antiviral therapy.

References

Akter, T., Ali, M. R., Faruquee, M. N. A., Bashir, M. S., Rahaman, S., Rahman, M. A., & Islam, S. (2024). A Comprehensive Study of Hepatitis B Infections in Bangladesh: Epidemiology, Risk Factors and Clinical-Laboratory Correlations. Viral Infections and Cancer Research, 1(1), 8426. https://doi.org/10.59429/vicr.v1i1.8426

Bashir, M. S., Sayem, M. A., Das, S. S., Das, N., Sultana, A., Rahman, M. M., Shamsuzzaman, M., Paul, P., Hossian, M., Uddin, M. K. M. (2025). "High Viral Load is a Risk Factor for Hepatocellular Carcinoma: Clinical and Laboratory Insights from a Cross-Sectional Study", Journal of Angiotherapy, 9(1),1-8,10219.  https://doi.org/10.25163/angiotherapy.9110219 

Czaja, A. J. (2014). Hepatic inflammation and progressive liver fibrosis in chronic liver disease. World Journal of Gastroenterology, 20(10), 2515. https://doi.org/10.3748/wjg.v20.i10.2515

D’souza, S., Lau, K. C., Coffin, C. S., & Patel, T. R. (2020a). Molecular mechanisms of viral hepatitis induced hepatocellular carcinoma. World Journal of Gastroenterology, 26(38), 5759–5783. https://doi.org/10.3748/wjg.v26.i38.5759

Ferenci, P., Fried, M., Labrecque, D., Bruix, J., Sherman, M., Omata, M., Heathcote, J., Piratsivuth, T., Kew, M. C., Ohnishi, K., & Reddy, R. K. (2010). Hepatocellular carcinoma (HCC): A global perspective. Journal of Clinical Gastroenterology, 44(4), 239–245. https://doi.org/10.1097/MCG.0b013e3181d46ef2

Gnyawali, B., Pusateri, A., Nickerson, A., Jalil, S., & Mumtaz, K. (2022). Epidemiologic and socioeconomic factors impacting hepatitis B virus and related hepatocellular carcinoma. World Journal of Gastroenterology, 28(29), 3793–3802. https://doi.org/10.3748/wjg.v28.i29.3793

Huang, M., Chen, H., Wang, H., Wang, X., Wang, D., Li, Y., ... & Ma, L. (2024). Global, regional, and national burden of liver cancer due to non-alcoholic steatohepatitis, 1990–2019: An analysis of the Global Burden of Disease Study. Research Square. https://doi.org/10.21203/rs.3.rs-4099455/v1

Islam, M. R., Yesmin, T., Prapty, A. N., Biswash, M. A. R., Rana, M. S., & Rashid, M. H. O. (2024). Natural environmental sources of resveratrol and its therapeutic role in cancer prevention. Australian Herbal Insight, 7(1), 1–11. https://doi.org/10.25163/ahi.719931

Kim, T. J., Sinn, D. H., Min, Y. W., Son, H. J., Kim, J. J., Chang, Y., ... & Ryu, S. (2017). A cohort study on Helicobacter pylori infection associated with nonalcoholic fatty liver disease. Journal of Gastroenterology, 52(11), 1201–1210. https://doi.org/10.1007/s00535-017-1337-y

Lala, V., Zubair, M., & Minter, D. (2023, July 30). Liver function tests. Treatment & Management | Point of Care. https://www.statpearls.com/point-of-care/20995/

Lodato, F. (2006). Hepatocellular carcinoma prevention: A worldwide emergence between the opulence of developed countries and the economic constraints of developing nations. World Journal of Gastroenterology, 12(45), 7239. https://doi.org/10.3748/wjg.v12.i45.7239

Mak, D., & Kramvis, A. (2021). Epidemiology and aetiology of hepatocellular carcinoma in Sub-Saharan Africa. Hepatoma Research. https://doi.org/10.20517/2394-5079.2021.15

Mantovani, A., Lando, M. G., Borella, N., Scoccia, E., Pecoraro, B., Gobbi, F., ... & Targher, G. (2024). Relationship between Helicobacter pylori infection and risk of metabolic dysfunction-associated steatotic liver disease: An updated meta-analysis. Liver International, 44(7), 1513–1525. https://doi.org/10.1111/liv.15925

Mantovani, A., Turino, T., Altomari, A., Lonardo, A., Zoppini, G., Valenti, L., Tilg, H., Byrne, C. D., & Targher, G. (2019). Association between Helicobacter pylori infection and risk of nonalcoholic fatty liver disease: An updated meta-analysis. Metabolism, 96, 56–65. https://doi.org/10.1016/j.metabol.2019.04.012

Mohammadi, M., Attar, A., Mohammadbeigi, M., Peymani, A., Bolori, S., & Fardsanei, F. (2023). The possible role of Helicobacter pylori in liver diseases. Archives of Microbiology, 205(8). https://doi.org/10.1007/s00203-023-03602-z

Naito, Y., & Yoshikawa, T. (2002). Molecular and cellular mechanisms involved in Helicobacter pylori-induced inflammation and oxidative stress. Free Radical Biology and Medicine, 33(3), 323–336. https://doi.org/10.1016/s0891-5849(02)00868-7

Okushin, K., Tsutsumi, T., Ikeuchi, K., Kado, A., Enooku, K., Fujinaga, H., ... & Koike, K. (2018). Helicobacter pylori infection and liver diseases: Epidemiology and insights into pathogenesis. World Journal of Gastroenterology, 24(32), 3617–3625. https://doi.org/10.3748/wjg.v24.i32.3617

Ozakyol, A. (2017b). Global Epidemiology of Hepatocellular Carcinoma (HCC Epidemiology). Journal of Gastrointestinal Cancer, 48(3), 238–240. https://doi.org/10.1007/s12029-017-9959-0

Rabelo-Gonçalves, E. M. (2015). Extragastric manifestations ofHelicobacter pyloriinfection: Possible role of bacterium in liver and pancreas diseases. World Journal of Hepatology, 7(30), 2968. https://doi.org/10.4254/wjh.v7.i30.2968

Rana, M. S., Bashir, M. S., Das, S. S., Hossain, M., & Barua, S. (2024). Biomarkers for hepatocellular carcinoma: Diagnosis, prognosis, and treatment response assessment – A systematic review. Journal of Primeasia, 5(1), 1–8. https://doi.org/10.25163/primeasia.519784

Rees, K. R., Sinha, K. P., & Spector, W. G. (1961). The pathogenesis of liver injury in carbon tetrachloride and thioacetamide poisoning. The Journal of Pathology, 81(1), 107–118. https://doi.org/10.1002/path.1700810113

Salam, R., Saliou, A., Bielle, F., et al. (2023). Cellular senescence in malignant cells promotes      tumor progression in mouse and patient glioblastoma. Nature Communications, 14, 36124. https://doi.org/10.1038/s41467-023-36124-9

Thrift, A. P. (2016). The epidemic of oesophageal carcinoma: Where are we now? Cancer Epidemiology, 41, 88–95. https://doi.org/10.1016/j.canep.2016.01.013

Tsukuma, H., Hiyama, T., Tanaka, S., Nakao, M., Yabuuchi, T., Kitamura, T., Nakanishi, K., Fujimoto, I., Inoue, A., Yamazaki, H., & Kawashima, T. (1993). Risk Factors for Hepatocellular Carcinoma among Patients with Chronic Liver Disease. New England Journal of Medicine, 328(25), 1797–1801. https://doi.org/10.1056/nejm199306243282501

Tufael, A., Kar, A., Or Rashid, M. H., Sunny, A. R., Raposo, A., Islam, M. S., Hussain, M. A., Hussen, M. A., Han, H., Coutinho, H. D. M., Ullah, M. S., & Rahman, M. M. (2024). Diagnostic efficacy of tumor biomarkers AFP, CA19-9, and CEA in hepatocellular carcinoma patients. Journal of Angiotherapy. https://doi.org/10.25163/angiotherapy.849513

Tufael, M., Rahman, M. M., Upadhye, V. J., Hossain, M. F., & Uddin, N. (2024). Combined biomarkers for early diagnosis of hepatocellular carcinoma. Journal of Angiotherapy, 8(5), 1–12. https://doi.org/10.25163/angiotherapy.859665

World Health Organization (WHO). (2023). Hepatitis B. https://www.who.int/news-room/fact-sheets/detail/hepatitis-b

Zheng, Z., & Wang, B. (2021). The Gut-Liver Axis in Health and Disease: The role of Gut Microbiota-Derived Signals in liver injury and Regeneration. Frontiers in Immunology, 12. https://doi.org/10.3389/fimmu.2021.775526

PDF
Full Text
Export Citation

View Dimensions


View Plumx



View Altmetric



64
Save
0
Citation
81
View
1
Share